One of the Internet’s basic truths that if you want to start an argument about terminal ballistics, ask people whether 9mm or .45 is the better caliber.
This article isn’t really about the debate between these calibers, but rather the science that supports people’s arguments. Today we’re starting the conversation about ballistics. Specifically, we’re talking about terminal ballistics. I’m also focusing primarily on how we got to the .223/5.56 and why it matters.
So why are we hitting this subject? Part of marksmanship is knowing not only how to aim and fire the rifle from a stable position, but what the bullet does during flight and on impact. Generally speaking, target shooters are less concerned about terminal ballistics than hunters or defensive-minded shooters.
Terminal ballistics is the study of how a projectile behaves when it hits its target and transfers its kinetic energy to the target. The bullet’s design, as well as its impact velocity, plays a huge role in how the energy is transferred.
Why This Matters
Target shooters don’t usually care about terminal ballistics compared to hunters or defensive shooters. For target shooters, once the bullet punches a hole or rings the gong, what happens next is immaterial. For that reason, they focus much more on what the bullet does in flight. Target shooters, especially at long range, favor designs that sacrifice terminal performance in favor of improved external ballistics.
However, if you need that bullet to not only hit the target but also cause enough tissue damage to “put the target down.” Then you care very much about terminal ballistics and should choose bullets that have favorable characteristics.
How Bullets [Don’t] Wound and Kill
This is a complicated topic and knowledge of how bullets wound targets evolved over the years. The ideas we operate under today are vastly different than what we thought 100 years ago, even though the technology hasn’t changed all that much.
I’ll get to the history of terminal ballistics in a second, but let’s just get something out of the way first. A bullet kills via two methods, and only two methods:
- Destroy something vital in the central nervous system (brain or spinal cord) so that electrical impulses are no longer generated or carried
- Drain enough blood from the body (via holes) so that oxygen cannot effectively circulate
The trick is in how we get to one of these two outcomes. So let’s talk history.
A Brief History of Wound Research
Let’s start in the 1870s with the work of Theodor Kocher. Born in Switzerland in 1841, Theodor Kocher rose to the rank of Colonel in the Swiss Militia. By 1872, he earned a position as a professor of surgery in Bern.
Kocher was particularly interested in the evolution of terminal ballistics as small arms transitioned from spherical musket balls to the more devastating Minié ball used during the American Civil War and other conflicts around the world.
He formalized several theories that would have lasting impacts. According to Dr. Kocher, the primary wounding channels were:
- Cavitation
- Deformation of the bullet which transferred more kinetic energy
- Bullets dragging surface contaminants into the wound
Let’s talk about each of these a little more, since they become very important later on.
Cavitation
Dr. Kocher was the first to formally test the hydraulic pressure theory. He demonstrated its merit by shooting sealed boxes filled with water. An article from the American College of Surgeons has a nice writeup on Kocher’s experiments.
A prevailing theory was that the increased tissue destruction from a conical Minié bullet came from the centrifugal force created by a twisted, or corkscrewed, path through tissue. Another explanation was that the bullet created a hydraulic pressure wave that violently tore tissue as it pushed aside, like the wake on a boat.
In short, Dr. Kocher believed that this pressure wave stretching and destroying tissue was a significant driver of rifle wounds in the body.
Bullet Deformation and Kinetic Energy
Another theory was that the elongated shape of the Minié ball led to bend and deform as it impacted. This deformation led to a greater kinetic energy transfer from the bullet in flight to the target.
Dr. Kocher worked with Col. Eduard Rubin of the Swiss Federal Ammunition Factory and created the first full-metal-jacket bullet. Dr. Kocher’s reasons stemmed from a desire to inflict less damage to the target by limiting the amount of bullet deformation. Colonel Rubin saw that these FMJ bullets had higher velocity, accuracy, and came at a lower weight.
It appeared to be a win/win.
This was actually the basis for the Hague Conventions, which enforced the use of FMJ bullets rather than expanding or soft-pointed “dum dum” bullets. The Americans and the British were the only two nations to vote against the measure.
The Americans had fought against the new style FMJ bullets during the Spanish-American War, and found the decreased lethality concerning (though appreciated the increased range and accuracy). The British, on the other hand, were frustrated by the lack of performance they perceived with the FMJ bullets while fighting in India.
The soft-point “dum dum” type of bullet they came to favor came from that conflict, where they snipped the tips off of FMJ bullets to expose the soft lead core and improve deformation.
Wound Contamination
This is less relevant to our discussion today since infection is something that causes problems later on and doesn’t immediately stop a threat.
Dr. Kocher proved that bullets, especially wider flatter ones, could drag harmful bacteria into a wound with them. This bacteria could come from anywhere, such as the skin or clothing, but that’s not why this was an important discovery.
At the time, most of the world assumed that the temperature of the bullet in flight was high enough to sterilize the surface of the projectile.
Moving on.
John Thompson
Louis LaGarde
The Le Garde-Thompson Tests
In 1904, the US Army undertook a study in handgun ballistics. They assigned the project to infantry Colonel John T. Thompson and Major Louis Anatole LaGarde of the Medical Corps.
To execute the tests, they shot several livestock animals with handguns in a variety of calibers. Some of these included the 9mm Luger, .45 ACP, .38 Long Colt, and .455 Webley.
Their conclusions were pretty straight forward:
The board was of the opinion that a bullet, which will have the shock effect and stopping effect at short ranges necessary for a military pistol or revolver, should have a caliber not less than .45. … None of the full-jacketed or metal-patch bullets (all of which were less than cal. . 45) showed the necessary shock effect or stopping power for a service weapon….
We are not acquainted with any bullet fired from a hand weapon that will stop a determined enemy when the projectile traverses soft parts alone. The requirements of such a bullet would need to have a sectional area like that of a 3-inch solid shot the recoil from which when used in hand weapons would be prohibitive. …
Finally the Board reached the conclusion that the only safeguard at close encounters is a well-directed rapid fire from nothing less than a .45-caliber weapon. With this end in view soldiers should be drilled to fire at moving targets until they have attained proficiency as marksmen.
Pay attention to the “shock effect” and “stopping power” statements. These are important later on.
The big takeaway from this experiment, according to a doctoral thesis by Nicholas Maiden for The University of Adelaide in Australia, was that the most important element of ballistic wounding was the permanent cavity. This is the tissue actually cut, crushed, and destroyed as the bullet passes through.
1930: The Work of R.H. Kent
Robert Kent of the Aberdeen’s Ballistic Research Laboratory published a little-known report in 1930. I’ve covered this particular report in before, so I won’t go into the gritty details gain.
The big take away is that everyone still believed that kinetic energy transfer was a primary terminal ballistics wounding mechanism for rifles. Kent theorized that a smaller and faster projectile could transfer an equivalent amount of kinetic energy to a target as a larger slower projectile.
If this was true, he surmised, then soldiers could carry more ammunition, lighter rifles, and be more effective on the battlefield than using traditional full-sized battle rifles.
If you’ve read my series on the Small Caliber High-Velocity program, then you know that this theory ultimately prevailed and resulted in the US Military adopting the M16 rifle chambered in a lightweight 55gr projectile known as M193.
But it all hinged on the kinetic energy transfer theory, which turns out to be incorrect.
Where it All Goes Wrong
From at least the late 1800s through the Vietnam War, everyone assumed the kinetic energy transfer model was correct. The “shock” of getting hit by a fast-moving projectile and absorbing its kinetic energy contributed to “stopping power” and made rifles more effective.
Early reports of experimental AR-15s from Vietnam talked about “devastating” damage and wounds. Dramatic photos publicized by the Swiss government showed graphic wounds created by the M16. They claimed it was inhumane to use in warfare since it did more damage than the 7.62 NATO cartridge that complied with the Swiss-derived Hague Conventions.
But where was the proof?
Enter Martin Fackler
Dr. Fackler served as a surgeon and a Colonel in the US Army Medical Corps. He worked as a field surgeon in Da Nang during Vietnam, and saw his fair share of bullet wounds. During this time, he noticed a disconnect between what was being said about rifle ballistics, particularly the 5.56, and what he actually observed in the field.
Dr. Fackler used a combination of logic and calibrated ballistic gel to suss out the real wounding mechanisms of rifle bullets. In a 1987 paper titled What’s Wrong With the Wound Ballistics Literature, and Why he dispels several of the commonly held beliefs and myths surrounding bullet wounds.
In 1980, I treated a soldier shot accidentally with an M16 M193 bullet from a distance of about ten feet. The bullet entered his left thigh and traveled obliquely upward. It exited after passing through about 11 inches of muscle. The man walked in to my clinic with no limp whatsoever: the entrance and exit holes were about 4 mm across, and punctate. X-ray films showed intact bones, no bullet fragments, and no evidence of significant tissue disruption caused by the bullet’s temporary cavity. The bullet path passed well lateral to the femoral vessels. He was back on duty in a few days. Devastating? Hardly.
The wound profile of the M193 bullet (page 29 of the Emergency War Surgery—NATO Handbook, GPO, Washington, D.C., 1988) shows that most often the bullet travels about five inches through flesh before beginning significant yaw. But about 15% of the time, it travels much farther than that before yawing—in which case it causes even milder wounds, if it missed bones, guts, lung, and major blood vessels. In my experience and research, at least as many M16 users in Vietnam concluded that it produced unacceptably minimal, rather than “massive”, wounds. After viewing the wound profile, recall that the Vietnamese were small people, and generally very slim. Many M16 bullets passed through their torsos traveling mostly point forward, and caused minimal damage. Most shots piercing an extremity, even in the heavier-built Americans, unless they hit bone, caused no more damage than a 22 caliber rimfire bullet.
Terminal Ballistics Myths
To start, Dr. Fackler emphasizes that there are only two mechanisms for wounding:
- Temporary cavitation: tissue stretched and temporarily displaced (as proposed by Theodor Koch)
- Permanent wound channel: tissue cut and crushed as the bullet passes
According to Dr. Fackler’s work, the theory of kinetic energy transfer is wrong. At worst it is totally irrelevant, at best it is an inconsistent wounding method and should not be relied upon.
Another myth surrounds the supersonic shockwave as the bullet penetrates. Many people assumed this was another wounding mechanic of SCHV projectiles. Fackler points out the logical fallacy here:
The lithotripter, a recent invention that uses this sonic pressure wave to break up kidney stones, generates a wave five times the amplitude of the one from a penetrating small arms projectile. Up to 2,000 of these waves are used in a single treatment session, with no damage to soft tissue surrounding the stone.
The “devastating” wounds documented by early AR-15s? There was nothing scientific about the reports. Nobody actually compared it to a the results of a full-powered rifle cartridge.
The “inhumane” findings of the Swiss government? They were intentionally misleading, using undersized pigs to show relative damage and selectively choosing the worst damage.
It was a politically-motivated stunt.
How Bullets Wound and Kill
This brings us to the modern age, where we know a lot more than we did when Dr. Kocher started his experiments in the late 1800s.
A bullet, any bullet, wounds effectively in only one of two ways:
Permanent Wound Channel
The permanent wound channel is the tissue cut, crushed, and displaced as the bullet passes. If this wound channel passes through some vital organ or tissue, like the brain or heart, then death occurs very quickly. The problem is that these vital areas are relatively small and hard to hit.
Alternatively, poking enough holes in other tissue allows blood to flow out of the circulatory system. If you hit a major blood vessel, like the aorta or femoral artery, then this blood loss occurs very quickly in as little as a single shot. But, it usually takes more than one hit to poke holes in all of these places.
That’s the reason for aiming at center mass. There’s a higher probability of hitting areas with a lot of blood flow. This is also why firing only a single shot is a bad idea in a life or death situation.
You fire until the threat stops.
If it's good enough for one, it's good enough for several. If you're going to shoot, you might as well give it "P" for plenty.
Temporary Cavitation
Temporary cavitation, as theorized by Dr. Koch, is a real phenomenon. However, the effect is overblown.
Imagine throwing a rock into a pond. The inertia of the rock penetrating the water causes a wave that ripples out. The same thing happens to live tissue as a bullet penetrates it. The inertia of the bullet temporarily moves tissue out of the way.
Since tissue is elastic, and mostly made of water, it quickly rebounds to its original position. Dr. Fackler points out that you really shouldn’t think of this as any more than blunt trauma. Most tissues in the body are stretchy and aren’t dramatically affected by this temporary cavity. But others, such as the liver and kidneys, are more susceptible.
Fragmentation
This is the area of terminal ballistics that Dr. Fackler is known for. He demonstrated that the worst wounds come from the fragmentation of the bullet.
If you recall my article on rifle twist rates, I talked about the aerodynamic forces working on a projectile in flight. The shape of a modern rifle bullet puts the center of pressure in front of the center of mass. That means that a bullet insufficiently stabilized is prone to tumble.
When a rifle bullet impacts soft tissue, dramatically reducing its velocity and spin rate, the bullet becomes unstable and begins tumbling. If the jacket is thin enough, the drag forces of traveling through tissue sideways tend to break the bullet apart. When this happens, pieces of the jacket and lead core fly off in various directions.
This is fragmentation.
This tumbling and fragmentation effect takes place some distance after the initial impact, typically 12-15 inches. With a human combatant, that means that fragmentation could happen inside the body.
Fragmentation and Terminal Ballistics
When you combine the possible shredding effect of fragmentation with the intense blunt trauma and stretching effect of temporary cavitation, you enable dramatic wound channels. Think of it like a rubber band that you nicked with a knife. What used to be easily stretchable will now rip and tear.
Shattered bone fragments can have the same effect, perforating surrounding stretchy tissue until it ruptures rather than stretches.
If both fragmentation and cavitation occur, you make the permanent wound channel much more intense.
But that’s a big if.
All rifle bullets do this to a degree, but smaller lighter bullets tend to do it better due to their construction. Even then, achieving this kind of synergistic effect isn’t 100% reliable. Which is why it’s so common to fire more than one shot.
Also, since fragmentation is related to the dramatic deceleration of the bullet, it helps to have the bullet moving at a higher velocity upon impact. This is why barrel length has an impact on the “optimal” effective range of the .223 projectile.
Putting it Together
When you look at all of this, you see why handguns are much less effective compared to rifles.
Handgun bullets don’t have the inertial power to trigger large temporary cavities, nor do they have the aerodynamic properties to fragment. Rifles do both.
A handgun relies entirely on shot placement and the permanent wound channel for damage. Expanding ammunition only slightly increases the diameter of that wound channel and the probability that you put a hole in something important..
Expanding hollow-point ammunition does not make up for poor marksmanship.
With rifles, soft-point expanding ammunition works well on animals because it causes both a larger temporary and a wider permanent wound channel. That’s assuming it doesn’t fragment, which we typically don’t want in a hunting round because it could ruin the meat.
A Word of Caution
Looking at all of this stuff is fun and interesting, at least to me, but it’s also based on a lot of theory and other people’s experiences. At the end of the day, seek out some professional training and advice on what works for you and your situation.
I’ve always liked this video of Clint Smith at Thunder Ranch laying down some “real talk.” Be aware of some harsh language in case you’ve got speakers turned up.
Wrapping Up
This is just the start of a longer discussion about ballistics.
The big takeaway here is that it’s an evolving science. I made a lot of use of Dr. Fackler’s work, but realize his work is based on his experiences. There are others in the field who disagree with him on the relevance of shock waves and other effects that Dr. Fackler discounted.
Stay safe, and I’ll see you next time.
“Expanding [handgun] ammunition increases the diameter of that wound channel and the probability that you put a hole in something important.”
Eh. The bullet doesn’t expand all *that* much – you either hit something or not. Real life is not like a shooting match where nicking a scoring ring counts as higher score. Where expanding handgun caliber bullets do bring a benefit, is reliable performance through both the target and any intermediate barriers, and *especially* reduction of overpenetration. Both very positive attributes for a service or self-defense bullet! (Also, both much harder to market than “our bullets make bigger holes”…)
You are correct, I don’t want to overstate the behavior of JHP handgun rounds. They aren’t magic. Shot placement is still the most important factor for effectiveness.
The issue of balancing barrier performance and overpenetration is also important, but something I want to talk about in another article.
Shot placement and as well as larger calibers matter. The larger the diameter; the more potential a vital is hit and more damage achieved.
Even with a vital hit, it can still take minutes for incapacitating, so the larger the better for speeding up the process.
“Real life is not like a shooting match where nicking a scoring ring counts as higher score.”
The hell it doesn’t, if you nick an artery, that’s the “winning shot” you need.
There have been documented shootings where tenths of an inch was almost that was needed for a fatal hit, and a larger diameter bullet would have achieved stopping the threat.
Thos why shooting the largest bore you can accurately fire is the best option.
“Born in Switzerland in 1941” ← 1841？
Ah, good catch. Thanks!
FWIW: No one involved in the Spanish-American War was issuing cartridges using spitzer projectiles. The contemporary Spanish-issue 7mm Mauser cartridge launched a roundnose 173-grain FMJ at ~2,300 fps.
The French were the first to adopt the sharp-pointed projectile concept in 1898 with their Balle D, but it remained a state secret at the time. The rest of the world did not follow until after the German adoption of the S Patrone in 1903. The Swiss didn’t adopt the spitzer until 1911 (GP11), and Spain waited until 1913 (Tipo S). If I remember correctly, Louis La Garde stated the first conflict to see the use of spitzers was the First Balkan War (1912-1913) with the Turkish 7.65x53mm Mauser.
Ah, you are correct. Thank you for the correction.
No worries, I’m always glad to help. Really, the performance of the .30-40 Krag issue cartridge was not that different from any of the other major power’s contemporary issue cartridge in the 7.5-8mm bore range. The perceived superiority of the 7mm Mauser in the Spanish-American War had a lot more to do with the battlefield tactics and terrain. The end result would have been much the same if the two opposing forces had swapped rifles. An offensive force is going to take casualties charging a prepared defensive position on a ridge line, especially when there is little cover or concealment between the two forces. It is inevitably worse when you assemble and hold your forces in clear view of the enemy’s defensive positions and their supporting artillery, as was done at San Juan Hill.
In related popular myths, the internal box magazine of the Winchester M1895 rifle was not likely designed for spitzer projectiles, as no one was yet producing them. If you look at Browning’s patents, the M1895 patent drawings show the use of a single-stack en-bloc clip, a design which Browning had also patented. Clearly, Winchester deleted this feature for the final commercial design.
You may wish to put a link to that PDF at the very beginning of your article and the very end of your article, so people can find it easier.
Hi Shorty, thanks for coming by! Which PDF are you referring to? I linked to several within the article.
Sorry, I meant the big one. Martin Facklers report on lies and misinformation in the ballistics community. Which, by the way, was a real eye-opener for me.
I’ve just found this site accidentally, but I’m glad I did. 🙂 As a hunter I can very much support your points in the article. I’m also a technical guy, who likes to understand how things work, and I’d like to improve my skills, so I always check the effects of my shots on animals, and what I’ve found yet is consistent with the facts you mentioned above: – Shockwave myth: I usually hunt animals (like deers) where headshot would destroy the trophy, or small critters (like fox) where a missed headshot from 100+ meters would be an unnecessary injury risk (rather than humane kill), so I usually avoid headshots in 90% of my shots. I aim mid-high front torso for lung shots. About 10-15% of the animals I shot there suddenly collapse the rest runs for 30-50 meters before the loss of blood incapacitates them. At first I also thought that the 10-15% sudden kill is caused by shockwaves, but after processing the animal I’ve checked and found that the projectile nicked or broke the spinal column in all that cases. (somewhat high shot) So suddenly incapacitating nervous system damage was also caused by the mechanical damage, rather than a hydraulic shockwave. – Temporal vs. permanent cavity: Temporal cavity as I’ve found really only helps if you hit some less elastic, or hard surfaced internal organs like liver, spleen, kidney. It effectively destroys this organs, causing massive bleeding which incapacitates the animal in a short time. But this organs… Read more »
Hey Andy, thanks for coming by and commenting! I’m glad you found the article helpful.
I’m also very glad you provided some real world examples of how terminal ballistics works for your hunting shots. Theory is one thing, but getting relatable examples is even better!
I’m glad I could add something. 🙂
One more thing on shockwave: I personally never hit an animal there (luckily) but there is the “spike shot” where you hit one of the spikes on the spinal column. (happens with wild boars sometimes) There in 100% of the cases the shot renders the animal unconscious, but it recovers in a short time (few minutes) and runs away. They usually fully recover without permanent damage. (we’ve found some animals with healed wound there) I think it works so that there you mechanically hit the spinal column which sends a real shockwave in the spinal fluid, which acts as a concussion. That makes a temporal effect (like you’d be punched in the face) but it usually is not deadly. So if there would be an incapacitating shockwave it would surely work in this cases as the bullet passes an inch from the central nervous system itself. Still there’s nothing permanent.
Thanks for a good article. I heard the story of the 45 ACP related to the relative in-effectiveness of the Army .38 against the drugged Moros as compared with the Army 45 Colt effectiveness. Bigger holes equal faster bleed out
I’m glad you enjoyed it! And yep, when it comes down to it, bigger holes in the plumbing will help drain the system.
>Go ahead and drop your answer in the comments now, just to get things started. And don’t say .40 or 10mm, you instigator.
Fair enough, .357 SIG.
Outside of the context of hunting, where preserving the meat is a priority, would the synergy effect you mention imply that fragmenting projectiles see preferred over expanding projectiles? If so, why is it that nearly all of the defensive ammunition on the market seems to have softpoint projectiles, or some other kind of expansion mechanism, rather than provisions for fragmenting?
You see self defense rounds as expanding because most marketed self defense rounds are handgun rounds. Fragmenting for self defense only works at rifle velocities. Handgun speeds doesnt create the same violent splash effect and yaw. Itsx a lot more then just fragmenting vs expanding, Matt coulda wrote 100s of pages on it. Even just in hunting you can use a 308 win and hunt coyotes with a 110gr v max or an elk with a 180 gr nosler partition. Two completely different bullets that are intended to wound differently.
The trouble here is that fragmentation is a relatively unreliable wounding mechanism. It’s a characteristic of a relatively light weight projectile moving at high velocities. Anything sold as primarily being fragmenting is going to carry a different connotation with it (i.e. frangible) and probably not gain a lot of market share.
In my opinion, we’re walking a fine balance with a lot modern loads because we demand the kind of accuracy, wind, and barrier performance that comes from sacrificing terminal performance. Bullet construction that provides great wounding characteristics (such as soft point) often suffer at the external ballistics piece.
For 223 specifically, which I based a lot of this article around, a lot of hunting bullet construction is geared towards small animals and wouldn’t have enough penetration on something larger.
It would seem clear that fragmenting pistol bullets are the proverbial right answer. It would appear FN was spot on when they developed the 5.7×28 and created the SS197SR. The SS198LF uses a nearly 1 inch long 27 grain bullet that in fact does tumble in soft tissue, and of course all this is viewable in any one of thousands of published videos. As for conventional pistol bullets, seems like we were doing it right back in the revolver era when most ammo was either lead bulleted or exposed lead nose jacketed. Certainly this article and the Thompson-Le Garde tests validated that lead bullets are much better stoppers and recommended jacketed bullets have a very thin jacket over the nose to allow it to deform. Today we have plated bullets which perform that duty nicely, as well as polymer coated lead that retains the malleable nature of lead alloy with a sealed “wrap.” There is a saying I coined that goes like this: A hit to a non-vital area with hollowpoint bullet is no more effective than a hit with a non-expanding bullet. A hit to a vital area with a non-expanding bullet is no less effective than a hit with a hollowpoint. Regardless of marketing hyperbole, gape-mouth hollowpoint bullets are NOT as reliable as rounded nose designs for feeding through semiautomatic pistols, and since it really is all about, “location, location, location” in terms of dropping an assailant, and the current dogma is to “shoot to stop the threat,”… Read more »
The most infamous failure of a bullet to stop and attacker was the 9mm Silvertip in the 1986 Miami shootout. The bullet performed EXACTLY as it was design – passed through the upper arm, missed the bone, into the chest, and directly to the heart where it came to rest outside the Pericardium – expanded, but completely out of sufficient energy to penetrate or in any other manner, damage the heart!
Had THAT bullet been a 115 to 124 grain non-expanding – even “round nose” design it would have pierced the Pericardial sac and entered the heart muscle, severing all sorts of nerve bundles, vessels, and heart muscle, and possibly passed through to create an exit hole on the other side!
That was a hollowpoint FAILURE, yet somehow seems to have spawned a profitable industry of marketing expanding and hollowpoint bullets!
>>38316724
This contradicts nothing of what I said. I think the confusion is in large part because of not understanding how bullets usually kill. Unless if hitting the heart or spinal column, which is pretty rare, it’s from the exit wounds causing blood loss. Even if a lung is punctured (do we know??), it can be grafted and patched reliably.
In summary, those exit wounds could easily seem like odd angles from the slight chance that Tim might’ve been laying down or otherwise contorted for the rounds.